![]() coli strains such as K1 indicate at least 6.5 million cases with attendant medical costs exceeding $2.5 billion annually in the United States alone. Escherichia coli K1-specific bacteriophage CUS-3 distribution and function in phase-variable capsular polysialic acid O acetylation Journal of Bacteriology. ![]() Minimal estimates of morbidity and economic costs associated with human infections caused by extraintestinal pathogenic E. 6 'ë ¿ þI Àôôzç ë ä :lp ðMêè੯²Ð0WÔî ïz:jÞÚ2SÎ éï úë a ¥ùÍqãoÊFU’Ê1-Q8ýåb¾0¿ -ØZ¤ OV µ NYÖÍ·vÜjeÕðV Ù YÆ» E õ-uñÜ K ã nQs ÎYB»OÚ &B ríà £òÅCn¡ Eä Æ å ÂçprĪ X¡èTäxËè ®Otõ´oi. King MR, Vimr RP, Steenbergen SM, Spanjaard L, Plunkett G, Blattner FR, Vimr ER. coli contingency locus shown to operate by a translational switch, further investigation of neuO should provide a better understanding of the invasive K1 pathotype. Acetylation is catalysed by the receptor‐modifying acetyl coenzyme‐A‐dependent O‐acetyltransferase encoded by neuO, a phase variable locus mapping near the integrase gene of the K1‐specific prophage, CUS‐3, which is inserted in argW at 53.1 map units. coli K1 strains undergo form (phase) variation involving loss or gain of O‐acetyl esters at carbon positions 7 or 9 of the individual sialic acid residues of the polysialic acid chains. Find Eric Vimr's phone number, address, and email on Spokeo, the leading people search directory for contact information and public records. A prominent virulence factor in these diseases is the polysialic acid capsular polysaccharide (K1 antigen), which is encoded by the kps/neu accretion domain inserted near pheV at 67 map units. VIMR Department of Veterinary Pathobiology Microbiology Division College of Veterinary Medicine University of Illinois at. Summary Escherichia coli K1 is part of a reservoir of adherent, invasive facultative pathogens responsible for a wide range of human and animal disease including sepsis, meningitis, urinary tract infection and inflammatory bowel syndrome. coli strains such as K1 indicate at least 6.5 million cases with attendant medical costs exceeding 2.5 billion US dollars annually in the United States alone.Mobile contingency locus controlling Escherichia coli K1 polysialic acid capsule acetylation Mobile contingency locus controlling Escherichia coli K1 polysialic acid capsule acetylation ![]() Minimal estimates of morbidity and economic costs associated with human infections caused by extraintestinal pathogenic E. Acetylation is catalysed by the receptor-modifying acetyl coenzyme-A-dependent O-acetyltransferase encoded by neuO, a phase variable locus mapping near the integrase gene of the K1-specific prophage, CUS-3, which is inserted in argW at 53.1 map units. ![]() #Eric vimr uiuc pdfcoli K1 strains undergo form (phase) variation involving loss or gain of O-acetyl esters at carbon positions 7 or 9 of the individual sialic acid residues of the polysialic acid chains. Sialic acid metabolisms dual function in Haemophilus influenzae Eric Vimr, Carol Lichtensteiger, Susan Steenbergen First published: Citations: 73 Sections PDF Tools Share Abstract Many bacterial commensals and pathogens use the sialic acids as carbon and nitrogen sources. Share this grant: : : Abstract Funding Institution Related projects Publications Comments Recent in Grantomics: Your institution vs. Search 24 grants from Eric Vimr Search grants from University of Illinois Urbana-Champaign. ![]() Vimr, Eric 2002 Watkins, Ruth 2001 Williamson, H. A prominent virulence factor in these diseases is the polysialic acid capsular polysaccharide (K1 antigen), which is encoded by the kps/neu accretion domain inserted near pheV at 67 map units. Vimr, Eric Ross University of Illinois Urbana-Champaign, Champaign, IL, United States. UNIVERSITY OF ILLINOIS URBANA-CHAMPAIGN SENATE Committee on Committees (Final - Action). Escherichia coli K1 is part of a reservoir of adherent, invasive facultative pathogens responsible for a wide range of human and animal disease including sepsis, meningitis, urinary tract infection and inflammatory bowel syndrome. ![]()
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